That figure is for viral myocarditis, which this arguably is, but there's no reason to think that rate is the same for a virus that's able to self replicate in perpetuity constantly producing cytotoxic virus fragments and for virus fragments that can only be produced until the original mRNA breaks down.
I'm saying there's no reason to equate something like hep-c constantly creating cytotoxic virus bits for the rest of someone's life and mrna that makes the cytotoxic virus bits for a few days. The former is continuous damage over time presumably increasing the mortality rate over time and the latter is a fixed amount of damage and you'd expect the mortality rate to drop over time.
There's existing research into how long mRNA lasts in cells. It varies but without a mechanism to replace it all of it will eventually break down and then it will be further broke down into peptides and then animo acids by recycling mechanisms in your cells.
That figure is also for clinically recorded cases. Myocarditis is one of those diseases that aren't easily diagnosed by blood test/imaging. MRI can do it but only if the heart is very significantly enlarged, but they generally don't do MRI scans without good reason. So they only usually catch myocarditis when it's bad enough to create symptoms like heart failure and they start looking for causes. Which means the vast majority of mild cases go undiagnosed.
That goes for vaccine induced myocarditis. The ones who end up in hospital and get diagnosed are at serious risk, but there's nothing to say those who got the vaccine and experienced some mild, subclinical inflammation are also at that same level of risk.
That figure is for viral myocarditis, which this arguably is, but there's no reason to think that rate is the same for a virus that's able to self replicate in perpetuity constantly producing cytotoxic virus fragments and for virus fragments that can only be produced until the original mRNA breaks down.
So are you saying it will be lower?
I'm saying there's no reason to equate something like hep-c constantly creating cytotoxic virus bits for the rest of someone's life and mrna that makes the cytotoxic virus bits for a few days. The former is continuous damage over time presumably increasing the mortality rate over time and the latter is a fixed amount of damage and you'd expect the mortality rate to drop over time.
Do we know that the mRNA wears off?
There's existing research into how long mRNA lasts in cells. It varies but without a mechanism to replace it all of it will eventually break down and then it will be further broke down into peptides and then animo acids by recycling mechanisms in your cells.
That figure is also for clinically recorded cases. Myocarditis is one of those diseases that aren't easily diagnosed by blood test/imaging. MRI can do it but only if the heart is very significantly enlarged, but they generally don't do MRI scans without good reason. So they only usually catch myocarditis when it's bad enough to create symptoms like heart failure and they start looking for causes. Which means the vast majority of mild cases go undiagnosed.
That goes for vaccine induced myocarditis. The ones who end up in hospital and get diagnosed are at serious risk, but there's nothing to say those who got the vaccine and experienced some mild, subclinical inflammation are also at that same level of risk.